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Shell Programming and Scripting
Find matches and write the data before it
Post 302728499 by Priyanka Chopra on Thursday 8th of November 2012 02:05:41 AM
11-08-2012
Hi
Thanks for reply.
but this time output file is completely blank!
but yeah, in the input second file there are more than 5 columns therefore, what I wanted is just write whatever is front of common match is present as it is and for sure in columns as input!
And, I checked in the previous output file there are not at all any columns rather entries of 5 columns are row wise..
and regarding tab seaparation entries are like this here each colur represent each column so in input file there are 8 columns.
so if FCGR2A is present in first file then output will be
Thanks for reply.
but this time output file is completely blank!
but yeah, in the input second file there are more than 5 columns therefore, what I wanted is just write whatever is front of common match is present as it is and for sure in columns as input!
And, I checked in the previous output file there are not at all any columns rather entries of 5 columns are row wise..
and regarding tab seaparation entries are like this here each colur represent each column so in input file there are 8 columns.
Quote:
FCGR2A FCGR2B FCGR2C EGFR FCGR3B C1R C1QA C1QB C1QC FCGR3A C1S FCGR1A Cetuximab Erbitux FCGR2A FCGR2B FCGR2C EGFR FCGR3B C1R C1QA C1QB C1QC FCGR3A C1S FCGR1A Cetuximab binds to the epidermal growth factor receptor (EGFr) on both normal and tumor cells. EGFr is over-expressed in many colorectal cancers. Cetuximab competitively inhibits the binding of epidermal growth factor (EGF) and TGF alpha, thereby reducing their effects on cell growth and metastatic spread. Epidermal growth factor receptor binding FAB. Cetuximab is composed of the Fv (variable; antigen-binding) regions of the 225 murine EGFr monoclonal antibody specific for the N-terminal portion of human EGFr with human IgG1 heavy and kappa light chain constant (framework) regions. For treatment of EGFR-expressing metastatic colorectal cancer in patients who are refractory to other irinotecan-based chemotherapy regimens. Cetuximab is also indicated for treatment of squamous cell carcinoma of the head and neck in conjucntion with radiation therapy. Used in the treatment of colorectal cancer, cetuximab binds specifically to the epidermal growth factor receptor (EGFr, HER1, c-ErbB-1) on both normal and tumor cells. EGFr is over-expressed in many colorectal cancers. Cetuximab competitively inhibits the binding of epidermal growth factor (EGF) and other ligands, such as transforming growth factor-alpha. Binding of cetuximab to the EGFr blocks phosphorylation and activation of receptor-associated kinases, resulting in inhibition of cell growth, induction of apoptosis, decreased matrix metalloproteinase secretion and reduced vascular endothelial growth factor production.
Quote:
FCGR2A etuximab Erbitux FCGR2A FCGR2B FCGR2C EGFR FCGR3B C1R C1QA C1QB C1QC FCGR3A C1S FCGR1A Cetuximab binds to the epidermal growth factor receptor (EGFr) on both normal and tumor cells. EGFr is over-expressed in many colorectal cancers. Cetuximab competitively inhibits the binding of epidermal growth factor (EGF) and TGF alpha, thereby reducing their effects on cell growth and metastatic spread. Epidermal growth factor receptor binding FAB. Cetuximab is composed of the Fv (variable; antigen-binding) regions of the 225 murine EGFr monoclonal antibody specific for the N-terminal portion of human EGFr with human IgG1 heavy and kappa light chain constant (framework) regions. For treatment of EGFR-expressing metastatic colorectal cancer in patients who are refractory to other irinotecan-based chemotherapy regimens. Cetuximab is also indicated for treatment of squamous cell carcinoma of the head and neck in conjucntion with radiation therapy. Used in the treatment of colorectal cancer, cetuximab binds specifically to the epidermal growth factor receptor (EGFr, HER1, c-ErbB-1) on both normal and tumor cells. EGFr is over-expressed in many colorectal cancers. Cetuximab competitively inhibits the binding of epidermal growth factor (EGF) and other ligands, such as transforming growth factor-alpha. Binding of cetuximab to the EGFr blocks phosphorylation and activation of receptor-associated kinases, resulting in inhibition of cell growth, induction of apoptosis, decreased matrix metalloproteinase secretion and reduced vascular endothelial growth factor production.
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